Nutrition Recommendations for Those who Consume Alcohol

However, obesity and the accumulation of subcutaneous adipose tissue are poor prognostic factors in patients with alcoholic cirrhosis, and nutritional therapy should be tailored to individual conditions [80]. Alcoholic cirrhosis results in a combination of undernutrition and overnutrition, which requires individualized management, including monitoring of nutritional status and nutritional guidance. Considering that enteral nutrition for liver failure contains approximately 200 kcal, overfeeding should be avoided by subtracting the amount of enteral nutrition from the total energy. The addition of LES without intervention may exacerbate glucose intolerance [81]. Energy intake is based on 25–35 kcal/kg (standard body weight)/day in the absence of glucose intolerance. In the presence of glucose intolerance, 25 kcal/kg/day and protein requirements are based on 1.0–1.5 g/kg/day (including BCAA preparations) in the absence of protein intolerance.

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multivitamin for alcoholics

In addition, the pathogenesis of liver damage, especially cirrhosis, further exacerbates nutritional disorders. Chronic alcohol abuse causes significant magnesium deficiencies [142]. Both total circulating multivitamin for alcoholics magnesium and ionized circulating magnesium levels are markedly decreased in chronic alcohol use disorders. Further, the normal response of the kidneys to hypomagnesemia is blunted [144,145].

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As a result, many individuals who misuse alcohol may become malnourished. In addition to conventional treatments and complete abstinence, Dr. Weil recommends the following two supplements to people coping with alcohol dependence. Alcohol interferes with nutrient absorption, damages the gastrointestinal tract, and impairs the liver’s ability to metabolize nutrients. Vitamin C can ease withdrawal by increasing the alcohol clearance rate9.

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Hyperhomocysteinemia induced by ethanol and linked to oxidative endoplasmic reticulum stress triggers apoptosis and increases lipid production [112]. Several research articles have linked alcohol-mediated oxidative stress and ethanol-inducible CYP2E1 with oxidative stress and their toxicity, both in in vitro and in vivo models. In effect, new pathophysiological focuses that could be used against ALD have been described using in vitro studies [29,30]. Nonetheless, hepatocytes’ antioxidant defense can counteract this damage through enzymatic as well as nonenzymatic mechanisms [31,32,33,34,35,36]. Recent clinical trials have examined the efficacy of numerous antioxidants, including S-adenosylmethionine (SAMe) and vitamin E. However, the conclusions drawn by these have been conflicting [31,32,37,38].

Does alcohol deplete vitamins and minerals?

They may also ask the patient questions about his or her drinking habits, using a standardized questionnaire, to identify psychological signs. Since many alcoholics are in denial about their problem, doctors may ask the patient’s permission to speak with family and friends as well. Nutritional strategies may be helpful for the treatment of alcoholics, including those with ALD. The most important treatment for ALD is abstinence from alcohol; however, alcohol is highly addictive, and thus, abstinence is often difficult. In particular, advanced ALD requires appropriate nutritional therapy according to the disease stage. There are various mechanisms of alcohol-induced hepatocellular damage [5].

multivitamin for alcoholics

The ingested alcohol is oxidized by alcohol dehydrogenase (ADH) and cytochrome P450 2E1 (CYP2E1) to acetaldehyde, which is converted to acetic acid by aldehyde dehydrogenase (ALDH). Reactive oxygen species (ROS) generated during alcohol metabolism inhibit antioxidant activity in hepatocytes [6]. Markers of oxidative stress include serum NADPH oxidase (NOX2) [7] and urinary 8-isoPGF2α [8]. Alcohol-induced changes in the gut microbiota may contribute to alcohol-induced oxidative stress and intestinal hyperpermeability [9].

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  • Many individuals who drink large quantities of alcohol are deficient in vitamin B1, or thiamine.
  • Here’s everything you need to know about using herbal supplements for alcohol withdrawal.
  • Given the frequency of deficiencies in B vitamins, zinc, and vitamin D, supplementation may be beneficial.
  • Vitamin E contributes to the histological improvement of liver injury in NASH [128].
  • Please seek professional care if you believe you may have a condition.

Edible plants such as fruits, vegetables, spices, grains, and teas have been reported to be effective against ALD via these various mechanisms [12]. Vitamins can only provide some protection against oxidative stress caused by alcohol. However, they cannot entirely prevent the adverse effects of chronic alcohol abuse on the brain. It’s also crucial for carbohydrate metabolism and energy production. Alcoholics are at risk for thiamine deficiency because alcohol inhibits the absorption of this vitamin.

“If we give thiamine to a whole bunch of people who don’t really need it, there is no harm done,” Shiling said. The total cost of the thiamine and supplies to administer it is $89 for a total of six 500-milligram treatments delivered three times a day for two days. After implementing the protocol, Shiling analyzed three months of data and found that of 57 patients admitted to CeDAR for alcohol use disorder, 22 received IV thiamine.

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